MealShape is backed by human clinical trials showing post-prandial blood sugar control.
As cinnamon extracts show potential to control blood sugar, HORN (La Mirada, CA) is securing North American distribution rights for a science-backed cinnamon extract.
Developed by Dialpha (Montferrier-sur-Lez, France), MealShape is a standardized Ceylon cinnamon extract from Madagascar. The ingredient developer has already performed studies on this cinnamon, which have found blood sugar benefits for both animals and humans. The cinnamon extract reportedly delays digestion and absorption of carbohydrates after a starchy meal, thereby preventing sugar crashes and insulin spikes. The companies say that by this action the cinnamon extract allows the body to convert food into energy instead of stored fat. Trials with MealShape have shown blood sugar reductions as great as 20%, and without insulin spikes, immediately following a meal. The ingredient is currently suitable for dietary supplements and functional foods.
MealShape is a “true cinnamon” extract, meaning that it is sourced from Cinnamomum verum and not a cheaper alternative from the same cinnamon family. Because of its cinnamon source, HORN emphasizes that its product is also low in coumarin, a toxin present in various cinnamon substitutes.
Sirio Pharma launches line of ready-to-market organic gummies and softgels called PureOrganix
August 26th 2024The new line is made up of three gummies and one softgel that are formulated to meet stringent EU-Organic certification criteria, and target women’s health, metabolic health, and heart health.
Recent review states that pentadecanoic acid may support cellular stability for better longevity
June 25th 2024According to the paper’s author, Stephanie Venn-Watson, DVM, MPH, deficiency in pentadecanoic acid of ≤0.2% total circulating fatty acids increases the risk of ferroptosis, which a type of cell death cause by the peroxidation of fragile fatty acids in cell membranes that combines with iron thus increasing reactive oxygen species, and disabling mitochondria.